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Outline of a computerized Chinese grammar enabling English and Swedish translation

This paper presents a computerized grammar which can analyze and generate a sample of pedagogical Chinese sentences, in particular those mentioned in the textbook Kinesiska är inte svårt (‘Chinese is not difficult’) by Göran Malmqvist 1974. Equivalent grammars for English and Swedish have also been constructed allowing translation between the three languages. The grammar model used is the Swetra g

Anhängarens makt i en ny religiös rörelse - The MOVE Organization

This article address the means by which early followers can exert influence within a new religious movement. The case study that is used to exemplify potential dynamics in such a situation is that of the MOVE Organization, a group founded in the early 1970s in Philadelphia, Pennsylvania by a man known as "John Africa". Instead of focusing on the founder, however, this article examines the role pla

Essays on Realized Volatility and Jumps

Financial markets sometimes generate significant discontinuities, so called jumps, triggered by large informational shocks and extreme events. In the last decade, there is an increasing interest in financial economics towards modeling these jumps which may have significant consequences for risk management, and portfolio allocation. This thesis extends the literature within these areas in several w

Radiative lifetimes for the 4p excited states of phosphorus and the oscillator strengths of solar lines

Radiative lifetimes for the levels of the 3s(2)3p(2)4p configuration in neutral phosphorus have been determined experimentally for the first time and also theoretically. Experiments were performed by using two-photon UV laser excitation and subsequent fluorescence detection in a laser-produced plasma. In relativistic Hartree-Fock calculations, the Cowan code extended to 42 configurations was used.

Therapeutic approaches to spinal and bulbar muscular atrophy.

Spinal and bulbar muscular atrophy is a hereditary motor neuron disease caused by trinucleotide repeat expansion in the androgen receptor gene. The disease mechanism probably involves a toxic gain of function in the mutant protein, because other mutations that cause a loss of androgen receptor function result in a different phenotype and the mutant protein is toxic in mouse models. In these models